Your enquiry0Mitochondrial research
RESEARCH USE ONLYNAD+, β-Nicotinamide adenine dinucleotide
NAD+ is the oxidized form of nicotinamide adenine dinucleotide, a cofactor connecting cellular redox reactions with NAD-consuming enzyme pathways. Select your format and quantity for pricing, batch availability and lead time.
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ABOUT THE MATERIAL
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NAD+ is the oxidized form of nicotinamide adenine dinucleotide, a cofactor connecting cellular redox reactions with NAD-consuming enzyme pathways.[Sources]
Reduction to NADH couples it to metabolic electron transfer. NAD+ also participates in the biology of CD38 and sirtuins, linking cofactor availability with mitochondrial and cellular metabolism.
NAD+ and NADH are a redox pair. Precursors such as nicotinamide riboside enter biosynthetic pathways before contributing to the NAD pool. Oxidation state and cofactor content are key analytical variables.
A CLOSER LOOK
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Camacho-Pereira and colleagues studied the NADase CD38 in age-associated NAD decline. The experiments linked CD38 activity to mitochondrial changes involving SIRT3 and to degradation of the precursor NMN. This provides an enzyme-pathway framework for NAD metabolism research.
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BEFORE YOU ENQUIRE
NAD+ and NADH are a redox pair. Precursors such as nicotinamide riboside enter biosynthetic pathways before contributing to the NAD pool. Oxidation state and cofactor content are key analytical variables.
Use the specification and available safety or stability information for the offered lot, form and packaging. Request written conditions before ordering; literature alone does not establish a universal temperature, shelf life or reconstitution procedure.
The displayed value is a catalogue specification. Assess the offered lot against its matching COA and method. Chromatographic purity, component content and biological activity are different measurements; confirm which results are available.
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SOURCES
Content updated
Camacho-Pereira J, Tarragó MG, Chini CCS, Nin V, Escande C, Warner GM, Puranik AS, Schoon RA, Reid JM, Galina A, Chini EN. 2016, DOI: 10.1016/j.cmet.2016.05.006
Mouse aging and cellular metabolism
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